公司提供的D-(-)水楊苷號：138-52-3 *，貨源充足。嚴格的生產(chǎn)質(zhì)量控制體系，包括：優(yōu)級純，分析純，化學純，試劑級，基準試劑，實驗純，教學試劑，高純試劑，色譜純，光譜純，電子純。各種包裝規(guī)格，并可提供包裝定制，咨詢訂購。
D-(-)水楊苷號：138-52-3   
英文名稱：D-(-)Salicin；2-(Hydroxymethyl)phenyl-beta-D-glucopyranoside    
其他名稱：柳醇；水楊素；水楊素糖；D(-)-水楊素；水楊甙；水楊糖   
號：138-52-3    
C13H18O7=286.28   
級別：BR   
含量：≥99.0% (HPLC)    
比旋光度：?62.5±2°（c=3% in H2O）   
熔點：199～202℃   
性狀(以下信息僅供參考)：無色結晶或白色粉末，有吸濕性。1g溶于23ml水、3ml沸水、90ml乙醇、30ml60℃時乙醇，溶于吡啶、堿和冰乙酸，幾乎不溶于乙迷和方。水溶液呈中性，而且味苦。相對密度1.434，有刺激性   
用途：本品僅供科研，不得用于其它用途。(以下用途僅供參考)測定β-糖苷酶的底物。   
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別    名  BEER; Cortical hyperostosis with syndactyly; Sclerosteosis; Sclerostin; SOST; SOST_HUMAN; VBCH.
濃    度  1mg/1ml
規(guī) 格  0.2ml/200μg
抗體來源  Rabbit 
克隆類型  polyclonal
交叉反應  Human, Mouse, Rat, Dog, Pig, Cow, Horse, Rabbit, Sheep 
產(chǎn)品類型  一抗   
研究領域  細胞生物 信號轉導 生長因子和激素 
蛋白分子量  predicted molecular weight: 21kDa
性    狀  Lyophilized or Liquid
免 疫 原  KLH conjugated synthetic peptide derived from human Sclerostin
亞    型  IgG
純化方法  affinity purified by Protein A
儲 存 液  Preservative: 15mM Sodium Azide, Constituents: 1% BSA, 0.01M PBS, pH 7.4
產(chǎn)品應用   WB=1:100-500  ELISA=1:500-1000  IHC-P=1:100-500  IHC-F=1:100-500  ICC=1:100-500  IF=1:100-500
（石蠟切片需做抗原修復）
 not yet tested in other applications.
 optimal dilutions/concentrations should be determined by the end user. 
保存條件  Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.
Important Note  This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications.
產(chǎn)品介紹 Negative regulator of bone growth.Sclerostin (SOST) is a bone morphogenetic protein (BMP) antagonist, leading to the activation of BMP signaling. It negatively regulates the formation of bone by repressing the differentiation and/or function of osteoblasts induced by BMPs. It has been shown that Sclerostin binds BMP-5, -6, and -7 with high affinity and BMP-2 and -4 with low affinity. The noggin-sclerostin protein complex represents a novel mechanism for the fine-tuning of BMP activity in bone homeostasis. Evidence is accumulating that one of the important mechanisms of bone regulation by sclerostin is the modulation of Wnt/Beta-catenin signaling. Sclerostin also rapidly activated ERK-1/2 MAPK signaling, indicating the involvement of additional signaling pathways
Function : Negative regulator of bone growth that acts through inhibition of Wnt signaling and bone formation.
Subunit : Interacts with LRP4 (via the extracellular domain); the interaction facilitates the inhibition of Wnt signaling. Interacts with LRP5 (via the first two YWTD-EGF repeat domains); the interaction inhibits Wnt-mediated signaling. Interacts with LRP6.
Subcellular Location : Secreted.
Tissue Specificity : Widely expressed at low levels with highest levels in bone, cartilage, kidney, liver, bone marrow and primary osteeoblasts differentiated for 21 days.
DISEASE : Defects in SOST are the cause of sclerosteosis (SOST) [MIM:269500]; also known as cortical hyperostosis with syndactyly. SOST is an autosomal recessive sclerosing bone dysplasia characterized by a generalized hyperostosis and sclerosis leading to a markedly thickened skull, with mandible, ribs, clavicles and all long bones also being affected. Due to narrowing of the foramina of the cranial nerves, facial nerve palsy, hearing loss and atrophy of the optic nerves can occur. Sclerosteosis is clinically and radiologically very similar to van Buchem disease, mainly differentiated by hand malformations and a large stature in sclerosteosis patients.
Note=A 52 kb deletion downstream of SOST results in SOST transcription suppression and is a cause of van Buchem disease (VBCH) [MIM:239100]; also known as hyperostosis corticalis generalisata. VBCH is an autosomal recessive sclerosing bone dysplasia characterized by endosteal hyperostosis of the mandible, skull, ribs, clavicles, and diaphyses of the long bones. Affected patients present a symmetrically increased thickness of bones, most frequently found as an enlarged jawbone, but also an enlargement of the skull, ribs, diaphysis of long bones, as well as tubular bones of hands and feet. The clinical consequence of increased thickness of the skull include facial nerve palsy causing hearing loss, visual problems, neurological pain, and, very rarely, blindness as a consequence of optic atrophy. Serum alkaline phosphatase levels are elevated.
Similarity : Belongs to the sclerostin family. Contains 1 CTCK (C-terminal cystine knot-like) domain.
Database links : UniProtKB/Swiss-Prot: Q9BQB4.1
英文名稱  Anti-Somatostatin
中文名稱  *抗體
別    名  Growth hormone release inhibiting factor; SMS_HUMAN; SMST; SST; Somatostatin-14; Somatostatin 14; Somatostatin14; somatostatin SS14; Somatostatin 28; Somatostatin28; Somatostatin-28; Somatostatin-28; somatostatin SS28.
濃    度  1mg/1ml
規(guī) 格  0.1ml/100μg  0.2ml/200μg
抗體來源  Rabbit 
克隆類型  polyclonal
交叉反應  Human, Mouse, Rat, Dog, Pig, Cow, Horse, Rabbit
產(chǎn)品類型  一抗   
研究領域  細胞生物 發(fā)育生物學 神經(jīng)生物學 干細胞 生長因子和激素 
蛋白分子量  predicted molecular weight: 3kDa
性    狀  Lyophilized or Liquid
免 疫 原  KLH conjugated synthetic peptide derived from human Somatostatin
亞    型  IgG
純化方法  affinity purified by Protein A
儲 存 液  Preservative: 15mM Sodium Azide, Constituents: 1% BSA, 0.01M PBS, pH 7.4